Your heart muscle (myocardium) is about the size of your clenched fist. It sits behind the breastbone and beats constantly, starting shortly after conception and continuing for more than seventy years in most Americans. Heart is really a remarkable pump: it pumps oxygenated blood to all the cells in the human body, including its own cells, bringing them needed nourishment and hormones, and it sends the used or deoxygenated blood back to the lungs so the cycle can begin again. Coronary artery disease (CAD), begins with a buildup of plaque in either the left or right coronary artery or their branches. This reduces the blood flow to the heart, but early on, this reduction is not severe enough to compromise heart muscle function or to produce any symptoms. Later, as the plaque enlarges and further reduces blood flow, blood flow interruption can reach the critical point where the heart muscle no longer gets adequate oxygen delivery when it is working vigorously (as when you exercise). Try heart disease first aid remedies to prevent or minimize the damage due to heart diseases.


A temporary or partial interruption in the supply of blood, injures the heart muscle and causes the chest pain known as angina. A prolonged or complete interruption kills myocardial cells and cause a heart attack. Generally, the more heart muscle a coronary artery feeds, the more devastating its failure is to the heart.

There are exceptions to this rule. For example, a relatively minor blockage can set off extreme electrical instability in the heart that can prove fatal because the heart muscle can no longer contract in a coordinated fashion. If the function of the electrical circuits could be restored in a timely fashion, through the use of drugs or a defibrillator, this injury might not even result in a detectable loss in muscle-pumping power.


Heart-shocking devices called defibrillators were once available only in hospitals and ambulances, but they‘re now popping up in public places. You can find these public versions, called automated external defibrillators, in airports, movie theaters, fitness centers, casinos, malls, office buildings, and elsewhere. The machines have easy-to understand instructions on them, and it’s basically just a matter of turning on the machine, attaching the pads to the victim’s bare chest—one on the upper part of the person’s right chest and the other on the left side near the armpit—waiting for the machine to analyze the heart rhythm, and pressing the shock or rescue button if the machine tells you to do so. Using these machines could save a life, as can performing cardiac resuscitation methods like CPR.


Most people think of a heart attack as something that happens quickly and causes someone to grab his or her chest and fall to the ground. That‘s actually a picture of cardiac arrest. A heart attack, as the term is commonly used today, generally means the blockage of an artery in the heart that kills some heart muscle. (Medically speaking; this is a myocardial infarction.) A heart attack usually gives some warning. Chest pain or other symptoms can prompt someone to get help before the blockage totally disrupts the heart‘s rhythm.

Cardiac arrest, though, strikes suddenly and out of the blue. Most cardiac arrests occur when the heart’s powerful lower chambers, the ventricles, start beating very fast or fast and chaotically (Ventricular fibrillation). Either one of these makes it impossible for the heart to pump blood to the body.

After just five seconds without blood circulation, a person passes out. In another few seconds, the lack of oxygen in the brain causes nerves to start firing, making the muscles twitch and the eyes roll back. Even that activity stops in less than a minute.


The chances of surviving a cardiac arrest fall about 10 percent for each minute the heart stays in ventricular fibrillation. Shock the heart back into a normal rhythm within two minutes, and the victim has an 80 percent chance of surviving. Deliver that shock after seven minutes—the average time it takes an emergency medical team to arrive in many cities—and the odds are less than 30 percent.

If someone near you goes into cardiac arrest, calling 911 is a must. CPR is also important because it keeps blood flowing to the brain and other vital organs. If there’s an automated external defibrillator nearby, use it following the instructions on the device.


What causes the blockages that set off these events? In a word, atherosclerosis. The term is a combination of two Greek words: athere, meaning pudding, and sclerosis, meaning hardening. The root words describe what happens in atherosclerosis: the artery walls become filled with soft, mushy deposits that eventually harden to make the artery stiff and narrow. Put simply, the arteries get clogged. The artery clogging seem straightforward, however, it’s a fairly complicated process.


There are four steps that occur in what is known as the coronary cascade to a heart attack. Though the reduced blood flow caused by the plaque formation taxes the heart, it doesn’t usually cause a heart attack. Heart attacks occur when the plaque ruptures. Plaque deposits team with inflammatory cells (particularly macrophages and other inflammatory cells called T-lymphocytes) as well as cholesterol. The more inflammatory cells and cholesterol— the thinner the cap that covers them— and the more unstable the plaque is. This sets the stage for disaster. T-cells slow the production of the fibrous materials that strengthen the cap (such as collagen), and macrophages produce enzymes that degrade collagen. This two-pronged attack degrades the cap until it breaks.

Although LDL is thought to be the major cholesterol-carrying culprit causing heart disease, there is still scientific controversy over the form LDL must take to cause atherosclerosis. The dominant view has been that LDL must first be oxidized to a more inflammatory form in order to cause serious artery wall damage. Oxidation may change the chemical structure of LDL molecules by breaking down large fat-containing molecular chains. Although there are literally thousands of studies that have suggested a role for oxidized LDL in causing heart disease, the use of antioxidants, such as vitamin E, has not resulted in any convincing decline in atherosclerosis in humans. The precise form LDL must take to set off atherosclerotic plaque formation remains a bit of a mystery, but the link between high LDL levels and coronary disease is firmly established.


The symptoms of heart attack differ between men and women and from person to person. Generally, men will report the following:

Pain or discomfort in the chest that radiates to the shoulder or arms, to the upper back near the shoulder blades, or to the neck or jaw
Uncomfortable pressure, tightness, fullness, or ache at the center of the chest
Shortness of breath, sweating, nausea, or dizziness
Women, on the other hand, usually report the following:
Pain in both arms or shoulders
Chest cramping or dull pain between the breasts
Shortness of breath
Feeling of indigestion
Lower abdominal pain
Severe fatigue (the least specific symptom, usually not caused by a heart attack)

If you or someone you know suffers a heart attack, or experience one or more of the symptoms listed above immediately call 911. Do not drive to hospital or medical center yourself. Once at the ER immediately approach the front desk and in a very sure and clear tone tell them that you are having a heart attack. Don’t waste any time in the waiting area at all.


Aspirin lowers platelets count in the blood, so that they do not form clots well. Some people who use aspirin occasionally may notice that they bleed longer from small cuts or may bruise more easily if they have taken aspirin recently. This minor annoyance can be a lifesaver, however, when platelets threaten to clump inside the coronary artery and block blood flow to the heart. If you can’t chew an aspirin, mash it up in a glass of water and drink it down. It‘s important not to take an aspirin whole; it can take too long for the body to break it down and absorb it.


If you’re alone, immediately call someone and tell him or her what’s going on. People don’t want to look foolish if it’s false alarm or don’t want to worry or bother others. Keep in mind: it’s much easier to live with embarrassment than with a damaged heart. So, if you feel like you’re having a heart attack and the symptoms last more than a few minutes, call 911 (or your local emergency number) sooner rather than later.


It’s easy to write off heart attack symptoms as something else. Chest pain can arise from stress- or activity-related angina. That hot, heavy feeling in the chest could be heartburn or gas. An ache in the left arm or jaw could be arthritis or the aftermath of snow shoveling.

Unfortunately, there’s no simple rule of thumb that separates a heart attack from a false alarm. And you’ll probably have a hard time being objective about it, which is why it’s so important to let a professional make an informed and unbiased judgment.

CAYENNE PEPPER EXTRACTsore throat remedies

There are stories of heart attack victims being pronounced dead, then being revived by a dose of Cayenne extract. While this is no substitute for CPR or a good doctor, it shows the power that many believe Cayenne Pepper holds over the body.

Dr. John Christopher N.D. declared: “In 35 years of practice, I have never on house calls lost one heart attack patient and the reason is, whenever I go in–if they are still breathing–I pour down them a cup of cayenne tea (a teaspoon of cayenne in a cup of hot water, and within minutes they are up and around).

The Cayenne pepper must be at least 90,000 heat units or 90,000(H.U.) to be able to stop a heart attack. If the cayenne is at least 90,000 H.U. and the person is still conscious, the recommendation is to mix 1 teaspoon of cayenne powder in a glass of warm water (this is essentially a “cayenne tea”), and give it to the person to drink.

If the person is unconscious then the recommendation is to use a cayenne tincture or extract, again of at least 90,000 H.U., and put a couple of full droppers underneath their tongue full strength.


Diet and life style changes along with the regular medication is a must for the survivors of a heart attack. You need to follow a heart-healthy diet, use strategies to control stress, and—it almost goes without saying—do not smoke.


All patients with coronary artery disease need to exercise and to discuss the progress of their exercise program regularly and in detail with their physicians. Aerobic exercise, such as walking, bicycling, or swimming, can help you lose weight or maintain a normal weight and increase the amount of work you can do with less strain on your heart.


Beta-blockers are among the most commonly used drugs for controlling interruptions in blood flow to the heart and high blood pressure, and for good reason—these drugs have been shown to improve survival rates after heart attacks, and they are especially effective at minimizing chest pain brought on by exercise.

People with asthma, heart failure, or diabetes should be cautious when taking beta-blockers because they could worsen these conditions. Despite these problems, beta—blockers are so effective in treating coronary artery disease that doctors often try them in patients with problems such as heart failure or diabetes because the benefits outweigh the risks.


Nitroglycerin and other nitrate compounds help prevent or stop ischemia in several ways. They relax the muscles in the walls of the blood vessels, causing arteries and veins to dilate. When the coronary arteries dilate in response to nitroglycerin, the heart’s blood supply increases. Nitrates also reduce the heart‘s work by lowering the body’s blood pressure and the pressure within the heart’s chambers. As a result, the heart requires less oxygen and places fewer demands on the coronary arteries.


ACE inhibitors are a class of blood pressure drugs that works by dilating blood vessels. In addition to controlling high blood pressure. ACE inhibitors have long been prescribed for people with heart failure. Recent studies have shown that these drugs also help people with coronary artery disease and those at high risk for developing it.


Like beta-blockers, calcium channel blockers control high blood pressure. Calcium channel dilate the coronary arteries. By doing so, they increase blood flow to the heart and cut its workload by reducing blood pressure and the force of the heart’s contractions.

In contrast to beta-blockers, there is thus far no evidence that calcium channel blockers improve survival after a heart attack in patients with coronary artery disease. But they are useful for patients who don’t get adequate relief from beta-blockers or nitrates. Calcium channel blockers are more effective than beta-blockers for preventing angina due to episodes of coronary artery constriction, often called coronary spasm.


Some fruits, can interfere with the metabolism of certain medications. If you’re on a calcium channel blocker or cholesterol lowering medication, talk to your doctor before eating grapefruit, pomelos, Seville oranges and any products made from these fruits, such as juices, marmalade, and compotes. If medication prevents you from enjoying these fruits, try others that are high in vitamin C, such as oranges and strawberries.


One of the pleasant surprises of the past two decades is the benefit of aspirin for patients with coronary artery disease. This common, inexpensive drug helps protect survivors of heart attack and stroke from subsequent heart attacks and death, and it even helps reduce the number of deaths that occur within the first hours following a heart attack.

Aspirin appears to work by preventing platelets from clumping together, which can block the blood flow to the heart. Randomized trials have provided clear evidence of aspirin’s value in both preventing and treating cardiovascular diseases. For high risk patients, regular aspirin use significantly decreased the risk of fatal and nonfatal strokes or heart attacks. A standard dose of aspirin to prevent heart attack is 81 mg per day.

Despite aspirin’s benefits, it also has some drawbacks. It can increase the risk for the less common form of stroke caused by bleeding into the brain, and it also makes significant gastrointestinal bleeding more likely. So the use of aspirin is recommended for people who already have heart disease or don’t yet have it but are at relatively high risk. When balancing the risk of heart disease versus aspirin‘s risks, the tipping point seems to be about 6 percent: for people with a ten-year heart disease risk of 6 percent or higher, the benefits of taking aspirin outweigh the harm. For people with a risk below 6 percent, they don’t. Using the 6 percent rule, an aspirin a day probably makes sense for the following people:

Anyone who has had a heart attack
Anyone diagnosed with coronary artery disease, peripheral artery disease, or a stroke or ministroke due to a blocked artery
Adults with diabetes, because this condition leads to heart disease
Adults with the “metabolic syndrome,” a combination of obesity, high blood pressure, and high levels of cholesterol and blood sugar
Healthy people with a 6 percent or greater chance of having a heart attack over the next ten years, including many men over age forty and women who have passed menopause
Although this list makes it look as if everyone should be taking aspirin, that’s certainly not the case. It isn’t a good option if you are prone to gastrointestinal bleeding or have had a hemorrhagic stroke, even if your heart attack risk is above 6 percent.

Garlic is a great natural home remedy that help lower your blood pressure and cholesterol along with many other wonderful health benefits such as helping with coronary heart disease, heart attack, and atherosclerosis.

This herb is used to protect against the early stages of heart disease, for pressure or tightness in chest, and for mild arrhythmia. It’s also useful for those recovering from a heart attack.
INDIAN GOOSEBERRYIndian gooseberry

Take 2-3 Kg fresh Indian Gooseberry and get it dry in shade ( no sunlight at all). Put them in a pot for 20 to 30 days for natural drying. After drying separate the seeds and ground the dried gooseberry to powder form. When it become powder mix equal quantity of sugar powder with it. Put it in a ceramic or plastic air tight pot. Take 6 gram (Two teaspoon) with fresh water in the morning, 30 minutes before breakfast. Use this powder for at least 2 months. If you want, you may take this powder for long period. It has no side effect.